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DESCRIBE <http://purl.uniprot.org/SHA-384/3383B0B8AA4CC469F6961E4DE235D8A18C8BD9A13A05CFFCBE0917E41AA093BD9AFD56C56C251EF13996DAC4FC631B7C>
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http://purl.uniprot.org/SHA-384/3383B0B8AA4CC469F6961E4DE235D8A18C8BD9A13A05CFFCBE0917E41AA093BD9AFD56C56C251EF13996DAC4FC631B7C
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/3383B0B8AA4CC469F6961E4DE235D8A18C8BD9A13A05CFFCBE0917E41AA093BD9AFD56C56C251EF13996DAC4FC631B7C
http://www.w3.org/2000/01/rdf-schema#comment
"established Neuro2a cells with edited GADD34 and ATF4/GADD34 genes and found that ATF4 acts as a proapoptotic factor but GADD34 depletion did not attenuate the expression of cleaved caspase-3 induced by tunicamycin treatment."
xsd:string
http://purl.uniprot.org/uniprot/#_92B1D72F967488D115B5C3E02B026FEB37450C5E257ABC748CB99E5D75A580E4E2CE69FD643AF4309DB4F3990530D011
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/3383B0B8AA4CC469F6961E4DE235D8A18C8BD9A13A05CFFCBE0917E41AA093BD9AFD56C56C251EF13996DAC4FC631B7C
http://purl.uniprot.org/uniprot/Q3TZI8
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3383B0B8AA4CC469F6961E4DE235D8A18C8BD9A13A05CFFCBE0917E41AA093BD9AFD56C56C251EF13996DAC4FC631B7C
http://purl.uniprot.org/uniprot/#_Q3TZI8-mappedCitation-28825160
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3383B0B8AA4CC469F6961E4DE235D8A18C8BD9A13A05CFFCBE0917E41AA093BD9AFD56C56C251EF13996DAC4FC631B7C