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DESCRIBE <http://purl.uniprot.org/SHA-384/34646FDE0F41946AEBDBEC50FC945324ACA3DA77D00E2CF6ADEAE7048219D35A50CB371947D53AB09B1BD5F99B646068>
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http://purl.uniprot.org/SHA-384/34646FDE0F41946AEBDBEC50FC945324ACA3DA77D00E2CF6ADEAE7048219D35A50CB371947D53AB09B1BD5F99B646068
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/34646FDE0F41946AEBDBEC50FC945324ACA3DA77D00E2CF6ADEAE7048219D35A50CB371947D53AB09B1BD5F99B646068
http://www.w3.org/2000/01/rdf-schema#comment
"activation of endogenous PKC-epsilon in hearts of diabetic epsilon-agonist mice promotes the survival phenotype attenuates markers of oxidative stress and inhibits the negative inotropic properties of chronic hyperglycemia"
xsd:string
http://purl.uniprot.org/uniprot/#_4F3F018F3C9A3948E5E104E88B01799E5F2970282B35F0363078F9D81926DB486C01C177F3C72249B5D27AFC25954753
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/34646FDE0F41946AEBDBEC50FC945324ACA3DA77D00E2CF6ADEAE7048219D35A50CB371947D53AB09B1BD5F99B646068
http://purl.uniprot.org/uniprot/Q8C944
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/34646FDE0F41946AEBDBEC50FC945324ACA3DA77D00E2CF6ADEAE7048219D35A50CB371947D53AB09B1BD5F99B646068
http://purl.uniprot.org/uniprot/#_Q8C944-mappedCitation-15894568
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/34646FDE0F41946AEBDBEC50FC945324ACA3DA77D00E2CF6ADEAE7048219D35A50CB371947D53AB09B1BD5F99B646068