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DESCRIBE <http://purl.uniprot.org/SHA-384/3599E6EB76353102CA4A6C158B99A7FF059F9D82E11AA395C760ACD3C401E0F2F95C523A9C4E324F1DCFC8BD9F4B0875>
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http://purl.uniprot.org/SHA-384/3599E6EB76353102CA4A6C158B99A7FF059F9D82E11AA395C760ACD3C401E0F2F95C523A9C4E324F1DCFC8BD9F4B0875
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/3599E6EB76353102CA4A6C158B99A7FF059F9D82E11AA395C760ACD3C401E0F2F95C523A9C4E324F1DCFC8BD9F4B0875
http://www.w3.org/2000/01/rdf-schema#comment
"Loss of crossbridge inhibition drives pathological cardiac hypertrophy in patients harboring the TPM1 E192K mutation."
xsd:string
http://purl.uniprot.org/uniprot/#_16E4A939D412B38C965B070693BFB69D0A980406174EF37F6DA7D0264C6ABA8C5B40A4217E7A5D93F3C9074251FD1CE2
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/3599E6EB76353102CA4A6C158B99A7FF059F9D82E11AA395C760ACD3C401E0F2F95C523A9C4E324F1DCFC8BD9F4B0875
http://purl.uniprot.org/uniprot/B7Z596
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3599E6EB76353102CA4A6C158B99A7FF059F9D82E11AA395C760ACD3C401E0F2F95C523A9C4E324F1DCFC8BD9F4B0875
http://purl.uniprot.org/uniprot/#_B7Z596-mappedCitation-34319370
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3599E6EB76353102CA4A6C158B99A7FF059F9D82E11AA395C760ACD3C401E0F2F95C523A9C4E324F1DCFC8BD9F4B0875