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DESCRIBE <http://purl.uniprot.org/SHA-384/3702BF5A8E5775FF6C0C4FE0C9EBA7E942B2A2759E866094D80B157174DEACC67F02213F7AE37431F608963790013C94>
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http://purl.uniprot.org/SHA-384/3702BF5A8E5775FF6C0C4FE0C9EBA7E942B2A2759E866094D80B157174DEACC67F02213F7AE37431F608963790013C94
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/3702BF5A8E5775FF6C0C4FE0C9EBA7E942B2A2759E866094D80B157174DEACC67F02213F7AE37431F608963790013C94
http://www.w3.org/2000/01/rdf-schema#comment
"Results show that amplified signaling of interleukin-17 receptor B (IL-17RB) and its ligand IL-17B promot tumorigenicity in breast cancer cells and impeded acinus formation in immortalized normal mammary epithelial cells."
xsd:string
http://purl.uniprot.org/uniprot/#_4E1441EB8241992BADB98620BF2682110C9BE470A649D41967B36DCB6508C76F2790BBC409C02EA8484AFE8009881889
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/3702BF5A8E5775FF6C0C4FE0C9EBA7E942B2A2759E866094D80B157174DEACC67F02213F7AE37431F608963790013C94
http://purl.uniprot.org/uniprot/Q9UHF5
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3702BF5A8E5775FF6C0C4FE0C9EBA7E942B2A2759E866094D80B157174DEACC67F02213F7AE37431F608963790013C94
http://purl.uniprot.org/uniprot/#_Q9UHF5-mappedCitation-23851503
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3702BF5A8E5775FF6C0C4FE0C9EBA7E942B2A2759E866094D80B157174DEACC67F02213F7AE37431F608963790013C94