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DESCRIBE <http://purl.uniprot.org/SHA-384/377B2CEFB96B04EEF71FF80EBFE69C78F3A83BB786E113631047292226A0E9F73296FEDFF48E662AF3B88536CF56293C>
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http://purl.uniprot.org/SHA-384/377B2CEFB96B04EEF71FF80EBFE69C78F3A83BB786E113631047292226A0E9F73296FEDFF48E662AF3B88536CF56293C
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/377B2CEFB96B04EEF71FF80EBFE69C78F3A83BB786E113631047292226A0E9F73296FEDFF48E662AF3B88536CF56293C
http://www.w3.org/2000/01/rdf-schema#comment
"Data demonstrate that the double mutations at the 409 and 410 sites in cardiac Na(+) channels induce inactivation-deficient sodium currents (INa) and that n-3 polyunsaturated fatty acids inhibit mutant I(Na)."
xsd:string
http://purl.uniprot.org/uniprot/#_7BF6CDF15325680154D7A777EE15408B97FC14B2D776F5CAE7191B8FF91E731C0C3206B2E00B5269B3A9B760976BA195
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/377B2CEFB96B04EEF71FF80EBFE69C78F3A83BB786E113631047292226A0E9F73296FEDFF48E662AF3B88536CF56293C
http://purl.uniprot.org/uniprot/A3EY21
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/377B2CEFB96B04EEF71FF80EBFE69C78F3A83BB786E113631047292226A0E9F73296FEDFF48E662AF3B88536CF56293C
http://purl.uniprot.org/uniprot/#_A3EY21-mappedCitation-16207794
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/377B2CEFB96B04EEF71FF80EBFE69C78F3A83BB786E113631047292226A0E9F73296FEDFF48E662AF3B88536CF56293C