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DESCRIBE <http://purl.uniprot.org/SHA-384/3890FB446BF5B923C63F88D035CD0272C7E93B8FD0A5DEFEB2BFCBD61FD51A3516F7ED8B0E28CBFE103E49B530964C96>
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http://purl.uniprot.org/SHA-384/3890FB446BF5B923C63F88D035CD0272C7E93B8FD0A5DEFEB2BFCBD61FD51A3516F7ED8B0E28CBFE103E49B530964C96
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/3890FB446BF5B923C63F88D035CD0272C7E93B8FD0A5DEFEB2BFCBD61FD51A3516F7ED8B0E28CBFE103E49B530964C96
http://www.w3.org/2000/01/rdf-schema#comment
"CCL5 modulates the innate inflammatory response acting through CCR1 to increase sepsis-induced lethality in wild-type mice in a cecal ligation and puncture model of septic peritonitis."
xsd:string
http://purl.uniprot.org/uniprot/#_22E6AF941AE3AF9739F2CDE54EBD2080A37DBE369D794F551296436334898F408B2C450C133AE5FA25DFABF6A9B2F6FF
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/3890FB446BF5B923C63F88D035CD0272C7E93B8FD0A5DEFEB2BFCBD61FD51A3516F7ED8B0E28CBFE103E49B530964C96
http://purl.uniprot.org/uniprot/Q5XZF2
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3890FB446BF5B923C63F88D035CD0272C7E93B8FD0A5DEFEB2BFCBD61FD51A3516F7ED8B0E28CBFE103E49B530964C96
http://purl.uniprot.org/uniprot/#_Q5XZF2-mappedCitation-15557190
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3890FB446BF5B923C63F88D035CD0272C7E93B8FD0A5DEFEB2BFCBD61FD51A3516F7ED8B0E28CBFE103E49B530964C96