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DESCRIBE <http://purl.uniprot.org/SHA-384/38CA2EB6B9AEC817473664BE2F407B9928DD1D9FF5FBE28686CD794BFF0CDB8B55EFE77ABBF6BD9776C1DA1C014A81AB>
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http://purl.uniprot.org/SHA-384/38CA2EB6B9AEC817473664BE2F407B9928DD1D9FF5FBE28686CD794BFF0CDB8B55EFE77ABBF6BD9776C1DA1C014A81AB
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/38CA2EB6B9AEC817473664BE2F407B9928DD1D9FF5FBE28686CD794BFF0CDB8B55EFE77ABBF6BD9776C1DA1C014A81AB
http://www.w3.org/2000/01/rdf-schema#comment
"Homozygous Cln3(delta ex7/8) transgenic mice represent the most appropriate disease model for studying the development of the pathogenetic events of juvenile neuronal ceroidlipofuscinoses."
xsd:string
http://purl.uniprot.org/uniprot/#_A768E09C919BFB944D686D62467DE7394480D7D6CB92E4B086BC3078EAFA31F86990F7A0E3AA4E2D3A1CCE03FF415F1E
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/38CA2EB6B9AEC817473664BE2F407B9928DD1D9FF5FBE28686CD794BFF0CDB8B55EFE77ABBF6BD9776C1DA1C014A81AB
http://purl.uniprot.org/uniprot/Q13286
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/38CA2EB6B9AEC817473664BE2F407B9928DD1D9FF5FBE28686CD794BFF0CDB8B55EFE77ABBF6BD9776C1DA1C014A81AB
http://purl.uniprot.org/uniprot/#_Q13286-mappedCitation-18265413
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/38CA2EB6B9AEC817473664BE2F407B9928DD1D9FF5FBE28686CD794BFF0CDB8B55EFE77ABBF6BD9776C1DA1C014A81AB