Results

RDF/XMLNTriplesTurtleShow queryShare
SubjectPredicateObject
http://purl.uniprot.org/SHA-384/3904015381F7FBD917934F764D25D93BBBEA63F76F6C43798E8E69AAC6C564F15E3DC7D1AD1D960DAC7B9267B5D8C251http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/3904015381F7FBD917934F764D25D93BBBEA63F76F6C43798E8E69AAC6C564F15E3DC7D1AD1D960DAC7B9267B5D8C251http://www.w3.org/2000/01/rdf-schema#comment"Luciferase reporter assay revealed that miR-1290 directly bound to the 3'-UTR of INPP4B; the mutated seed sites in miR-1290 abrogated this effect. Double knockdown of INPP4B and miR-1290 promoted CRC cell proliferation suggesting miR-1290 promoted CRC cell proliferation by targeting INPP4B."xsd:string
http://purl.uniprot.org/uniprot/#_D22220D5CF992DA94C10CAC17E043A08EF082A172772AC4FA1351692323F53F4F2C86B569D38882C107BC0FE1F438BC2http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/3904015381F7FBD917934F764D25D93BBBEA63F76F6C43798E8E69AAC6C564F15E3DC7D1AD1D960DAC7B9267B5D8C251
http://purl.uniprot.org/uniprot/E7EQN9http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/3904015381F7FBD917934F764D25D93BBBEA63F76F6C43798E8E69AAC6C564F15E3DC7D1AD1D960DAC7B9267B5D8C251
http://purl.uniprot.org/uniprot/#_E7EQN9-mappedCitation-28915933http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/3904015381F7FBD917934F764D25D93BBBEA63F76F6C43798E8E69AAC6C564F15E3DC7D1AD1D960DAC7B9267B5D8C251