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DESCRIBE <http://purl.uniprot.org/SHA-384/3A5F7109E1839B5ACD42BFCF438F6A71B7F99E57CC7492FCD672929D471AEF7B4A693C4D93038541F0C6A4ED303B40CC>
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http://purl.uniprot.org/SHA-384/3A5F7109E1839B5ACD42BFCF438F6A71B7F99E57CC7492FCD672929D471AEF7B4A693C4D93038541F0C6A4ED303B40CC
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/3A5F7109E1839B5ACD42BFCF438F6A71B7F99E57CC7492FCD672929D471AEF7B4A693C4D93038541F0C6A4ED303B40CC
http://www.w3.org/2000/01/rdf-schema#comment
"These results support a molecular mechanism for myotonia in myotonic dystrophy type 1 in which a reduction in both the number of functional sarcolemmal ClC-1 and maximal channel open probability."
xsd:string
http://purl.uniprot.org/uniprot/#_6B67DE48764F442805CDEAE1FCCD2EF8E648603AD0ADA9FD5355360DFA73889CE54479F5137F8AA0DD53D3606A048A7F
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/3A5F7109E1839B5ACD42BFCF438F6A71B7F99E57CC7492FCD672929D471AEF7B4A693C4D93038541F0C6A4ED303B40CC
http://purl.uniprot.org/uniprot/Q8K3N0
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3A5F7109E1839B5ACD42BFCF438F6A71B7F99E57CC7492FCD672929D471AEF7B4A693C4D93038541F0C6A4ED303B40CC
http://purl.uniprot.org/uniprot/#_Q8K3N0-mappedCitation-17158949
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3A5F7109E1839B5ACD42BFCF438F6A71B7F99E57CC7492FCD672929D471AEF7B4A693C4D93038541F0C6A4ED303B40CC