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DESCRIBE <http://purl.uniprot.org/SHA-384/3EBF043DD967E3ED28A40DE6A42C67231F0125C8D183EF39D4763F7B3CB418847E2F35EBEAAE86FBE0988C7B2335C63D>
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http://purl.uniprot.org/SHA-384/3EBF043DD967E3ED28A40DE6A42C67231F0125C8D183EF39D4763F7B3CB418847E2F35EBEAAE86FBE0988C7B2335C63D
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/3EBF043DD967E3ED28A40DE6A42C67231F0125C8D183EF39D4763F7B3CB418847E2F35EBEAAE86FBE0988C7B2335C63D
http://www.w3.org/2000/01/rdf-schema#comment
"splenocytes and macrophages rendered tolerant to TLR2 TLR4 and TLR9 ligands showed enhanced HIV-1 p24 production possibly reflecting a mechanism used by the virus to escape the effects of microbial-induced tolerance during infection."
xsd:string
http://purl.uniprot.org/uniprot/#_09CEB6663EF5092ABD04BD38A8B9F1A2E630A492FDE3C8747ABCFC3DE7A6604D7194A6C24F896FB11B410172836F623A
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/3EBF043DD967E3ED28A40DE6A42C67231F0125C8D183EF39D4763F7B3CB418847E2F35EBEAAE86FBE0988C7B2335C63D
http://purl.uniprot.org/uniprot/Q77YG1
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3EBF043DD967E3ED28A40DE6A42C67231F0125C8D183EF39D4763F7B3CB418847E2F35EBEAAE86FBE0988C7B2335C63D
http://purl.uniprot.org/uniprot/#_Q77YG1-mappedCitation-14657211
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3EBF043DD967E3ED28A40DE6A42C67231F0125C8D183EF39D4763F7B3CB418847E2F35EBEAAE86FBE0988C7B2335C63D