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DESCRIBE <http://purl.uniprot.org/SHA-384/3F6C528B69006842B758719CBE90A1F5EB1854E666EB4F548ABF2D2C262EDDCD2FFA625E815D54ABBD8B7A89FCC0B914>
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http://purl.uniprot.org/SHA-384/3F6C528B69006842B758719CBE90A1F5EB1854E666EB4F548ABF2D2C262EDDCD2FFA625E815D54ABBD8B7A89FCC0B914
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/3F6C528B69006842B758719CBE90A1F5EB1854E666EB4F548ABF2D2C262EDDCD2FFA625E815D54ABBD8B7A89FCC0B914
http://www.w3.org/2000/01/rdf-schema#comment
"These studies implicate CHIP as a stress-dependent regulator of Daxx that counters the pro-apoptotic influence of Daxx in the cell."
xsd:string
http://purl.uniprot.org/uniprot/#_7AAC79D7AA74FD5D27970E1F927A570BEEBBE46C5D98C21E210027209A09C330A32B0543AFA04F9AE7EAAED4BC87115C
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/3F6C528B69006842B758719CBE90A1F5EB1854E666EB4F548ABF2D2C262EDDCD2FFA625E815D54ABBD8B7A89FCC0B914
http://purl.uniprot.org/uniprot/Q5XLB0
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3F6C528B69006842B758719CBE90A1F5EB1854E666EB4F548ABF2D2C262EDDCD2FFA625E815D54ABBD8B7A89FCC0B914
http://purl.uniprot.org/uniprot/#_Q5XLB0-mappedCitation-19465479
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/3F6C528B69006842B758719CBE90A1F5EB1854E666EB4F548ABF2D2C262EDDCD2FFA625E815D54ABBD8B7A89FCC0B914