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DESCRIBE <http://purl.uniprot.org/SHA-384/40DD7F381F3EBE8F7C0E56ABAD18BFDB1D98761F0A7BC7AEFA1C33517E220FC99FA16C4DD1BFBD4136363F79755AA255>
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http://purl.uniprot.org/SHA-384/40DD7F381F3EBE8F7C0E56ABAD18BFDB1D98761F0A7BC7AEFA1C33517E220FC99FA16C4DD1BFBD4136363F79755AA255
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/40DD7F381F3EBE8F7C0E56ABAD18BFDB1D98761F0A7BC7AEFA1C33517E220FC99FA16C4DD1BFBD4136363F79755AA255
http://www.w3.org/2000/01/rdf-schema#comment
"On the basis of these data FOXD3 is a potent repressor of DCLK1-S expression in normal cells; loss of FOXD3 in hCCCs/hCRCs allows upregulation of DCLK1-S imparting a potent invasive potential to the cells"
xsd:string
http://purl.uniprot.org/uniprot/#_6E14D511F5AFDD846F849257B17FF769CA12F3278BBE3C0E491E80DF183DD6A828AEAADAD37FEF9F8DD33128754CD636
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/40DD7F381F3EBE8F7C0E56ABAD18BFDB1D98761F0A7BC7AEFA1C33517E220FC99FA16C4DD1BFBD4136363F79755AA255
http://purl.uniprot.org/uniprot/B7Z5K4
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/40DD7F381F3EBE8F7C0E56ABAD18BFDB1D98761F0A7BC7AEFA1C33517E220FC99FA16C4DD1BFBD4136363F79755AA255
http://purl.uniprot.org/uniprot/#_B7Z5K4-mappedCitation-28851816
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/40DD7F381F3EBE8F7C0E56ABAD18BFDB1D98761F0A7BC7AEFA1C33517E220FC99FA16C4DD1BFBD4136363F79755AA255