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DESCRIBE <http://purl.uniprot.org/SHA-384/41FF0328B4C6FF3FC97B5FA40662E03CFC0F2BD18F56DF9DA4424B144A4C7FFB67B74A4B5B231D99EA3868981D6ED316>
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http://purl.uniprot.org/SHA-384/41FF0328B4C6FF3FC97B5FA40662E03CFC0F2BD18F56DF9DA4424B144A4C7FFB67B74A4B5B231D99EA3868981D6ED316
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/41FF0328B4C6FF3FC97B5FA40662E03CFC0F2BD18F56DF9DA4424B144A4C7FFB67B74A4B5B231D99EA3868981D6ED316
http://www.w3.org/2000/01/rdf-schema#comment
"Thus these findings demonstrate a tumor-suppressive function of DYNC1I1 and uncover new mechanistic insights into SK2 regulation which may have implications in targeting this enzyme as a therapeutic strategy in glioblastoma"
xsd:string
http://purl.uniprot.org/uniprot/#_EE02086375428591D6449776EA84B786DB0EE3FB888DE0212DFF9D5105DA5225E855FE6BA1B75C0CB97C7D8C860DA4D8
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/41FF0328B4C6FF3FC97B5FA40662E03CFC0F2BD18F56DF9DA4424B144A4C7FFB67B74A4B5B231D99EA3868981D6ED316
http://purl.uniprot.org/uniprot/A4D1I7
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/41FF0328B4C6FF3FC97B5FA40662E03CFC0F2BD18F56DF9DA4424B144A4C7FFB67B74A4B5B231D99EA3868981D6ED316
http://purl.uniprot.org/uniprot/#_A4D1I7-mappedCitation-30250299
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/41FF0328B4C6FF3FC97B5FA40662E03CFC0F2BD18F56DF9DA4424B144A4C7FFB67B74A4B5B231D99EA3868981D6ED316