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DESCRIBE <http://purl.uniprot.org/SHA-384/425FB0CC8DCEA3B58CCB4B77F620BC12BD3C01B1B53A7CE3BC248880C269AD9C7AD59E875A952A0505B1C04F6BEA89A2>
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http://purl.uniprot.org/SHA-384/425FB0CC8DCEA3B58CCB4B77F620BC12BD3C01B1B53A7CE3BC248880C269AD9C7AD59E875A952A0505B1C04F6BEA89A2
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/425FB0CC8DCEA3B58CCB4B77F620BC12BD3C01B1B53A7CE3BC248880C269AD9C7AD59E875A952A0505B1C04F6BEA89A2
http://www.w3.org/2000/01/rdf-schema#comment
"A modest genetic increase in ACE impairs myocardial tolerance to ischemia. ACE level plays a critical role in cardiac ischemia through both kinin and angiotensin mediated mechanisms."
xsd:string
http://purl.uniprot.org/uniprot/#_6C653B108D8AA2E3686BF6BF2009A92AFEED90BA5CF30ED8F368B4EF4AC61962BF9B92932428CA5FC38E2F5949D8D6C7
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/425FB0CC8DCEA3B58CCB4B77F620BC12BD3C01B1B53A7CE3BC248880C269AD9C7AD59E875A952A0505B1C04F6BEA89A2
http://purl.uniprot.org/uniprot/P09470
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/425FB0CC8DCEA3B58CCB4B77F620BC12BD3C01B1B53A7CE3BC248880C269AD9C7AD59E875A952A0505B1C04F6BEA89A2
http://purl.uniprot.org/uniprot/#_P09470-mappedCitation-20667972
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/425FB0CC8DCEA3B58CCB4B77F620BC12BD3C01B1B53A7CE3BC248880C269AD9C7AD59E875A952A0505B1C04F6BEA89A2