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DESCRIBE <http://purl.uniprot.org/SHA-384/42A452F0352587BFBD7446DD9A5E0DC308752EA025F83F3871F249F76DAE7741C00D0B9C546E135E5E7DCBAE110D17B1>
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http://purl.uniprot.org/SHA-384/42A452F0352587BFBD7446DD9A5E0DC308752EA025F83F3871F249F76DAE7741C00D0B9C546E135E5E7DCBAE110D17B1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/42A452F0352587BFBD7446DD9A5E0DC308752EA025F83F3871F249F76DAE7741C00D0B9C546E135E5E7DCBAE110D17B1
http://www.w3.org/2000/01/rdf-schema#comment
"Data show that data provide a novel mechanism whereby CaMKII may regulate the proteasome in neurons to facilitate remodeling of synaptic connections through protein degradation."
xsd:string
http://purl.uniprot.org/uniprot/#_F27A285806D8A1BDF71A06D408C1BFAF4EE3891CE59B1C4173F7456FA32525D2E854E980967012E91349B4252C4112B6
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/42A452F0352587BFBD7446DD9A5E0DC308752EA025F83F3871F249F76DAE7741C00D0B9C546E135E5E7DCBAE110D17B1
http://purl.uniprot.org/uniprot/Q9NSM5
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/42A452F0352587BFBD7446DD9A5E0DC308752EA025F83F3871F249F76DAE7741C00D0B9C546E135E5E7DCBAE110D17B1
http://purl.uniprot.org/uniprot/#_Q9NSM5-mappedCitation-19638347
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/42A452F0352587BFBD7446DD9A5E0DC308752EA025F83F3871F249F76DAE7741C00D0B9C546E135E5E7DCBAE110D17B1