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DESCRIBE <http://purl.uniprot.org/SHA-384/42DB444D142B9BD2B8AAF91EA59CF1383350C062C8AD93AD3AED05D93BE1C60978B75C97314AB524E4E17B2B4DAF3B41>
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http://purl.uniprot.org/SHA-384/42DB444D142B9BD2B8AAF91EA59CF1383350C062C8AD93AD3AED05D93BE1C60978B75C97314AB524E4E17B2B4DAF3B41
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/42DB444D142B9BD2B8AAF91EA59CF1383350C062C8AD93AD3AED05D93BE1C60978B75C97314AB524E4E17B2B4DAF3B41
http://www.w3.org/2000/01/rdf-schema#comment
"Data show that genetic deficiency of calpastatin (CS) augmented Abeta amyloidosis tau phosphorylation microgliosis and somatodendritic dystrophy and increased mortality in amyloid precursor protein (APP) transgenic mice."
xsd:string
http://purl.uniprot.org/uniprot/#_BB975F6F8CD8B4A49802E59B37B3E0F482955E9240377B6A1A229A2740EEFBE08E7A002EAB9444649F6BE4AA6B8A7032
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/42DB444D142B9BD2B8AAF91EA59CF1383350C062C8AD93AD3AED05D93BE1C60978B75C97314AB524E4E17B2B4DAF3B41
http://purl.uniprot.org/uniprot/Q8CE80
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/42DB444D142B9BD2B8AAF91EA59CF1383350C062C8AD93AD3AED05D93BE1C60978B75C97314AB524E4E17B2B4DAF3B41
http://purl.uniprot.org/uniprot/#_Q8CE80-mappedCitation-22173972
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/42DB444D142B9BD2B8AAF91EA59CF1383350C062C8AD93AD3AED05D93BE1C60978B75C97314AB524E4E17B2B4DAF3B41