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DESCRIBE <http://purl.uniprot.org/SHA-384/44A1B960EE750766B5E1C90298633DD2F79F673347737CBE38850F50E1E75C23B1AE0EBF5549A21C5963DBBF28D13285>
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http://purl.uniprot.org/SHA-384/44A1B960EE750766B5E1C90298633DD2F79F673347737CBE38850F50E1E75C23B1AE0EBF5549A21C5963DBBF28D13285
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/44A1B960EE750766B5E1C90298633DD2F79F673347737CBE38850F50E1E75C23B1AE0EBF5549A21C5963DBBF28D13285
http://www.w3.org/2000/01/rdf-schema#comment
"These results suggest that SRF is critical for HCC to acquire a mesenchymal phenotype which leads to resistance against a sorafenib-mediated apoptotic effect."
xsd:string
http://purl.uniprot.org/uniprot/#_DF33794F7CBC0B28C40EC3E07D42002F371496A372AF93FD8B6193634442153865FC21EF396E8389AFE6A41D54997111
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/44A1B960EE750766B5E1C90298633DD2F79F673347737CBE38850F50E1E75C23B1AE0EBF5549A21C5963DBBF28D13285
http://purl.uniprot.org/uniprot/B4DU24
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/44A1B960EE750766B5E1C90298633DD2F79F673347737CBE38850F50E1E75C23B1AE0EBF5549A21C5963DBBF28D13285
http://purl.uniprot.org/uniprot/#_B4DU24-mappedCitation-24173109
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/44A1B960EE750766B5E1C90298633DD2F79F673347737CBE38850F50E1E75C23B1AE0EBF5549A21C5963DBBF28D13285