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DESCRIBE <http://purl.uniprot.org/SHA-384/44FFEFE457FA8FD5A224B120066A873AC1689C5721B92B53B01CD45DE3C29058F5E530ACB327C80901B33780A457A0CB>
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http://purl.uniprot.org/SHA-384/44FFEFE457FA8FD5A224B120066A873AC1689C5721B92B53B01CD45DE3C29058F5E530ACB327C80901B33780A457A0CB
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/44FFEFE457FA8FD5A224B120066A873AC1689C5721B92B53B01CD45DE3C29058F5E530ACB327C80901B33780A457A0CB
http://www.w3.org/2000/01/rdf-schema#comment
"Suggest that the increase in Slc26a7 expression caused by deletion of AE1 represents an adaptive response indicating that Slc26a7 contributes to the regulation of acid-base balance by the kidney."
xsd:string
http://purl.uniprot.org/uniprot/#_34575C7196731E1CEDA21A4104EF609155AC4D08B0817907F174B09481BCE8386D1B2A8FA3EB9914D808720197759A6C
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/44FFEFE457FA8FD5A224B120066A873AC1689C5721B92B53B01CD45DE3C29058F5E530ACB327C80901B33780A457A0CB
http://purl.uniprot.org/uniprot/P04919
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/44FFEFE457FA8FD5A224B120066A873AC1689C5721B92B53B01CD45DE3C29058F5E530ACB327C80901B33780A457A0CB
http://purl.uniprot.org/uniprot/#_P04919-mappedCitation-18663336
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/44FFEFE457FA8FD5A224B120066A873AC1689C5721B92B53B01CD45DE3C29058F5E530ACB327C80901B33780A457A0CB