"Influenza-infected STAT2-/- mice had increased morbidity viral burden and inflammation compared to wild-type mice. Absence of STAT2 was associated with increased accumulation of M1 M2 and M1/M2 co-expressing macrophages during influenza-bacterial super-infection. Results suggest that the STAT2 signaling is involved in suppressing macrophage activation and bacterial control during influenza-bacterial super-infection."xsd:string