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DESCRIBE <http://purl.uniprot.org/SHA-384/4E4656EA4EB6FFD937FB269F38B5717C8DA842A0D526F6BE3DFC53BA8044B067CE1BDB71B1225FD4B6BE77F6E47E38F4>
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http://purl.uniprot.org/SHA-384/4E4656EA4EB6FFD937FB269F38B5717C8DA842A0D526F6BE3DFC53BA8044B067CE1BDB71B1225FD4B6BE77F6E47E38F4
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/4E4656EA4EB6FFD937FB269F38B5717C8DA842A0D526F6BE3DFC53BA8044B067CE1BDB71B1225FD4B6BE77F6E47E38F4
http://www.w3.org/2000/01/rdf-schema#comment
"Data indicate that ephrin-A1 regulates cardiac valve development making ephrin-A1-deficient mice a novel model for congenital heart defects."
xsd:string
http://purl.uniprot.org/uniprot/#_8BBD688C98EF7AFDA173514DC98FEF252648B604497C57E6C4B1A8358CDA5047A696AFBE7B49FD4D1EBF2D1DEAD307F7
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/4E4656EA4EB6FFD937FB269F38B5717C8DA842A0D526F6BE3DFC53BA8044B067CE1BDB71B1225FD4B6BE77F6E47E38F4
http://purl.uniprot.org/uniprot/D3YTT5
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/4E4656EA4EB6FFD937FB269F38B5717C8DA842A0D526F6BE3DFC53BA8044B067CE1BDB71B1225FD4B6BE77F6E47E38F4
http://purl.uniprot.org/uniprot/#_D3YTT5-mappedCitation-20960543
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/4E4656EA4EB6FFD937FB269F38B5717C8DA842A0D526F6BE3DFC53BA8044B067CE1BDB71B1225FD4B6BE77F6E47E38F4