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DESCRIBE <http://purl.uniprot.org/SHA-384/534AF2F45B9FBEC35601BB05EBE7A76E43C8E090AF6CC3AE5F3EF6217134B621BEE9060EFBE9D21DE6529847AB1FF2D7>
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http://purl.uniprot.org/SHA-384/534AF2F45B9FBEC35601BB05EBE7A76E43C8E090AF6CC3AE5F3EF6217134B621BEE9060EFBE9D21DE6529847AB1FF2D7
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/534AF2F45B9FBEC35601BB05EBE7A76E43C8E090AF6CC3AE5F3EF6217134B621BEE9060EFBE9D21DE6529847AB1FF2D7
http://www.w3.org/2000/01/rdf-schema#comment
"Chalepensin was a substrate and a mechanism-based inhibitor of human CYP2A6; oral administration of chalepensin to mice reduced hepatic coumarin 7-hydroxylation activity ex vivo"
xsd:string
http://purl.uniprot.org/uniprot/#_FBC55F97B453F8038E972CA5BEB7A30180B1018D05031F1ECA5604EB713A0557185321D942ED8249EC5BFE8FA69A8BF4
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/534AF2F45B9FBEC35601BB05EBE7A76E43C8E090AF6CC3AE5F3EF6217134B621BEE9060EFBE9D21DE6529847AB1FF2D7
http://purl.uniprot.org/uniprot/P11509
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/534AF2F45B9FBEC35601BB05EBE7A76E43C8E090AF6CC3AE5F3EF6217134B621BEE9060EFBE9D21DE6529847AB1FF2D7
http://purl.uniprot.org/uniprot/#_P11509-mappedCitation-21418183
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/534AF2F45B9FBEC35601BB05EBE7A76E43C8E090AF6CC3AE5F3EF6217134B621BEE9060EFBE9D21DE6529847AB1FF2D7