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DESCRIBE <http://purl.uniprot.org/SHA-384/53F92D4D2DD62AA9B8773426A528D0F34BF34C4A93CFB617773F91260969147DF7AA8BEDA07165630C7BCC86F6F9774B>
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http://purl.uniprot.org/SHA-384/53F92D4D2DD62AA9B8773426A528D0F34BF34C4A93CFB617773F91260969147DF7AA8BEDA07165630C7BCC86F6F9774B
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/53F92D4D2DD62AA9B8773426A528D0F34BF34C4A93CFB617773F91260969147DF7AA8BEDA07165630C7BCC86F6F9774B
http://www.w3.org/2000/01/rdf-schema#comment
"Activation of Cdc42 GTPase through p210(Bcr-Abl) tyrosine kinase signaling in CML cells contributes to defects in SDF-1alpha-chemotactic response due to desensitization of the actin polarization signal required for directional migration."
xsd:string
http://purl.uniprot.org/uniprot/#_C28E63A889A4D7EB5F13CFB3F42924F91307FF49C2E3DA682FBDD678DF387F627AF2AB747B33CF2B4E01FBAAD736DBD5
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/53F92D4D2DD62AA9B8773426A528D0F34BF34C4A93CFB617773F91260969147DF7AA8BEDA07165630C7BCC86F6F9774B
http://purl.uniprot.org/uniprot/Q6QJE4
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/53F92D4D2DD62AA9B8773426A528D0F34BF34C4A93CFB617773F91260969147DF7AA8BEDA07165630C7BCC86F6F9774B
http://purl.uniprot.org/uniprot/#_Q6QJE4-mappedCitation-19718053
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/53F92D4D2DD62AA9B8773426A528D0F34BF34C4A93CFB617773F91260969147DF7AA8BEDA07165630C7BCC86F6F9774B