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http://purl.uniprot.org/SHA-384/5452AC4222BAE018708779C8BCBFCBA4D7C86113B45EAF954A4D23FBCCFB5B2FB09EFC7E68D591C785E77792D6AF00ABhttp://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/5452AC4222BAE018708779C8BCBFCBA4D7C86113B45EAF954A4D23FBCCFB5B2FB09EFC7E68D591C785E77792D6AF00ABhttp://www.w3.org/2000/01/rdf-schema#comment"In an animal endometriosis model unlike wild-type controls Klf10(-/-) animals developed cystic lesions with massive immune infiltrate and minimal peri-lesional fibrosis. The Klf10(-/-) disease progression phenotype also contrasted with prolific fibrosis and minimal immune cell infiltration seen in Klf11(-/-) animals."xsd:string
http://purl.uniprot.org/uniprot/#_C8A021BD2CC2E8124707C4DBBAF4131B68D9A8210AE4E0910B07C5A9D3EDBAB42BFF7D31CCE3574863D4D21116017589http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/5452AC4222BAE018708779C8BCBFCBA4D7C86113B45EAF954A4D23FBCCFB5B2FB09EFC7E68D591C785E77792D6AF00AB
http://purl.uniprot.org/uniprot/Q61596http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/5452AC4222BAE018708779C8BCBFCBA4D7C86113B45EAF954A4D23FBCCFB5B2FB09EFC7E68D591C785E77792D6AF00AB
http://purl.uniprot.org/uniprot/#_Q61596-mappedCitation-27488034http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/5452AC4222BAE018708779C8BCBFCBA4D7C86113B45EAF954A4D23FBCCFB5B2FB09EFC7E68D591C785E77792D6AF00AB