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http://purl.uniprot.org/SHA-384/54D5827F2A6FB8866CCAC37F8C723B04FCDA350B56B26A2F4E93BAE952821DD8CA04A45026E5E787C2B548193E82E1F8http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/54D5827F2A6FB8866CCAC37F8C723B04FCDA350B56B26A2F4E93BAE952821DD8CA04A45026E5E787C2B548193E82E1F8http://www.w3.org/2000/01/rdf-schema#comment"MAO inhibition prevented both mast cell degranulation and altered collagen deposition thereby normalizing diastolic function. These results elucidate the mechanisms underlying MAO-induced damage in diabetic cardiomyopathy and provide novel evidence for the role of MAOs in inflammation and inter-organelle communication."xsd:string
http://purl.uniprot.org/uniprot/#_7D9D307A54114F08508B897A87FAD41B510FE67B7843B20BD4E3093DB48038EC5A84A89715B27A2B33A0D3878681EA95http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/54D5827F2A6FB8866CCAC37F8C723B04FCDA350B56B26A2F4E93BAE952821DD8CA04A45026E5E787C2B548193E82E1F8
http://purl.uniprot.org/uniprot/Q3UJ53http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/54D5827F2A6FB8866CCAC37F8C723B04FCDA350B56B26A2F4E93BAE952821DD8CA04A45026E5E787C2B548193E82E1F8
http://purl.uniprot.org/uniprot/#_Q3UJ53-mappedCitation-29459772http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/54D5827F2A6FB8866CCAC37F8C723B04FCDA350B56B26A2F4E93BAE952821DD8CA04A45026E5E787C2B548193E82E1F8