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DESCRIBE <http://purl.uniprot.org/SHA-384/56B7C7B99C24B267E764989D798A947BE98B0B91CBFF564DCFC6A9C41489C83A4BD936FC2E414C12E8EC240DC7B5F0BF>
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http://purl.uniprot.org/SHA-384/56B7C7B99C24B267E764989D798A947BE98B0B91CBFF564DCFC6A9C41489C83A4BD936FC2E414C12E8EC240DC7B5F0BF
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/56B7C7B99C24B267E764989D798A947BE98B0B91CBFF564DCFC6A9C41489C83A4BD936FC2E414C12E8EC240DC7B5F0BF
http://www.w3.org/2000/01/rdf-schema#comment
"results show that a defect in the ability of insulin to activate Akt-2 and -3 may explain the impaired insulin-stimulated glucose transport in insulin resistance"
xsd:string
http://purl.uniprot.org/uniprot/#_F458568EA2810F5B05158588908B754B1F223796A914FB9E38062CDB95355012A46CA72719A59D94156E89496E2B3A62
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/56B7C7B99C24B267E764989D798A947BE98B0B91CBFF564DCFC6A9C41489C83A4BD936FC2E414C12E8EC240DC7B5F0BF
http://purl.uniprot.org/uniprot/B4DG79
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/56B7C7B99C24B267E764989D798A947BE98B0B91CBFF564DCFC6A9C41489C83A4BD936FC2E414C12E8EC240DC7B5F0BF
http://purl.uniprot.org/uniprot/#_B4DG79-mappedCitation-12663464
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/56B7C7B99C24B267E764989D798A947BE98B0B91CBFF564DCFC6A9C41489C83A4BD936FC2E414C12E8EC240DC7B5F0BF