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DESCRIBE <http://purl.uniprot.org/SHA-384/56FF43C547A82EF97ECA37BF29DEDD890FE01026AC4217DDE19BA7678E8B5F6C3EB8406E082AB73704C6F2C0284D3985>
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http://purl.uniprot.org/SHA-384/56FF43C547A82EF97ECA37BF29DEDD890FE01026AC4217DDE19BA7678E8B5F6C3EB8406E082AB73704C6F2C0284D3985
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/56FF43C547A82EF97ECA37BF29DEDD890FE01026AC4217DDE19BA7678E8B5F6C3EB8406E082AB73704C6F2C0284D3985
http://www.w3.org/2000/01/rdf-schema#comment
"That the CLCN7 mutations provoke a phenotype as severe as the one caused by TCIRG1 loss of function suggests the affected residues to be crucial for the function of the ClC-7 chloride channel or chloride/proton-exchanger"
xsd:string
http://purl.uniprot.org/uniprot/#_C1BF6FB3A9ED69BC76AB6D4015E0BB3A5BF29DBC1D4DFE719842B75B488B3214DD83D98BFAA38793B1D191A2935566DC
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/56FF43C547A82EF97ECA37BF29DEDD890FE01026AC4217DDE19BA7678E8B5F6C3EB8406E082AB73704C6F2C0284D3985
http://purl.uniprot.org/uniprot/B3KUD9
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/56FF43C547A82EF97ECA37BF29DEDD890FE01026AC4217DDE19BA7678E8B5F6C3EB8406E082AB73704C6F2C0284D3985
http://purl.uniprot.org/uniprot/#_B3KUD9-mappedCitation-20424301
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/56FF43C547A82EF97ECA37BF29DEDD890FE01026AC4217DDE19BA7678E8B5F6C3EB8406E082AB73704C6F2C0284D3985