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DESCRIBE <http://purl.uniprot.org/SHA-384/585BB21A26EAA96651FBB7ABCA3D94CF349EE3A41FDD0CE773EB87A6237A58A06019D3BD5A9C1229C3A85D9DBF0DE0F4>
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http://purl.uniprot.org/SHA-384/585BB21A26EAA96651FBB7ABCA3D94CF349EE3A41FDD0CE773EB87A6237A58A06019D3BD5A9C1229C3A85D9DBF0DE0F4
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/585BB21A26EAA96651FBB7ABCA3D94CF349EE3A41FDD0CE773EB87A6237A58A06019D3BD5A9C1229C3A85D9DBF0DE0F4
http://www.w3.org/2000/01/rdf-schema#comment
"Knockdown of TFAP2C or RET inhibited activation of ERK and AKT in MCF-7 cells. Knockdown of TFAP2C which controls ER (estrogen receptor) and RET had a greater effect on cell growth than either RET or ER alone."
xsd:string
http://purl.uniprot.org/uniprot/#_395365DFFC6116B2B5645307049463CF6EE7604CCBE0C0A2F293E7C39ED9716800AAF7780BF686C66B4CF8BD9327C1FC
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/585BB21A26EAA96651FBB7ABCA3D94CF349EE3A41FDD0CE773EB87A6237A58A06019D3BD5A9C1229C3A85D9DBF0DE0F4
http://purl.uniprot.org/uniprot/Q92754
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/585BB21A26EAA96651FBB7ABCA3D94CF349EE3A41FDD0CE773EB87A6237A58A06019D3BD5A9C1229C3A85D9DBF0DE0F4
http://purl.uniprot.org/uniprot/#_Q92754-mappedCitation-24045439
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/585BB21A26EAA96651FBB7ABCA3D94CF349EE3A41FDD0CE773EB87A6237A58A06019D3BD5A9C1229C3A85D9DBF0DE0F4