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DESCRIBE <http://purl.uniprot.org/SHA-384/58F6C4239DCB8FC5918375F444EB716E7BD4B8367C598C1801973E1B27C5BF07B833BA3311D6688B9AB4C5866AD872E3>
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http://purl.uniprot.org/SHA-384/58F6C4239DCB8FC5918375F444EB716E7BD4B8367C598C1801973E1B27C5BF07B833BA3311D6688B9AB4C5866AD872E3
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/58F6C4239DCB8FC5918375F444EB716E7BD4B8367C598C1801973E1B27C5BF07B833BA3311D6688B9AB4C5866AD872E3
http://www.w3.org/2000/01/rdf-schema#comment
"These results identify dAlk as an upstream activator of dNf1-regulated Ras signaling responsible for several dNf1 defects and they implicate human Alk as a potential therapeutic target in NF1"
xsd:string
http://purl.uniprot.org/uniprot/#_09D553F25F906B974BE0A1638EFE6219C91C7402A5B7FE4F3A1BC6EEDB93B792EF9F0B650F626EAB47B8800A69E71DDE
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/58F6C4239DCB8FC5918375F444EB716E7BD4B8367C598C1801973E1B27C5BF07B833BA3311D6688B9AB4C5866AD872E3
http://purl.uniprot.org/uniprot/Q59DT9
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/58F6C4239DCB8FC5918375F444EB716E7BD4B8367C598C1801973E1B27C5BF07B833BA3311D6688B9AB4C5866AD872E3
http://purl.uniprot.org/uniprot/#_Q59DT9-mappedCitation-21949657
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/58F6C4239DCB8FC5918375F444EB716E7BD4B8367C598C1801973E1B27C5BF07B833BA3311D6688B9AB4C5866AD872E3