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DESCRIBE <http://purl.uniprot.org/SHA-384/58FB6A344EDB6567F9328DD815094DF2AB090D21F26B7E9106FADC47F7742E2652E41C04F014535A256755DF8D7AE069>
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http://purl.uniprot.org/SHA-384/58FB6A344EDB6567F9328DD815094DF2AB090D21F26B7E9106FADC47F7742E2652E41C04F014535A256755DF8D7AE069
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/58FB6A344EDB6567F9328DD815094DF2AB090D21F26B7E9106FADC47F7742E2652E41C04F014535A256755DF8D7AE069
http://www.w3.org/2000/01/rdf-schema#comment
"Overexpression of mitochondrial Trx2 attenuates the intracellular ROS accumulation and ATP production. Most interestingly mitochondrial Trx2 may be involved in the cardiac hypertrophy signaling of diabetes."
xsd:string
http://purl.uniprot.org/uniprot/#_DAA62C149A30458B67CC3195BD56E5EC379691460D538DA3A9ECBFD1CA8F7841764C95FA217384AC47EE9713EF42B508
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/58FB6A344EDB6567F9328DD815094DF2AB090D21F26B7E9106FADC47F7742E2652E41C04F014535A256755DF8D7AE069
http://purl.uniprot.org/uniprot/Q99757
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/58FB6A344EDB6567F9328DD815094DF2AB090D21F26B7E9106FADC47F7742E2652E41C04F014535A256755DF8D7AE069
http://purl.uniprot.org/uniprot/#_Q99757-mappedCitation-28914755
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/58FB6A344EDB6567F9328DD815094DF2AB090D21F26B7E9106FADC47F7742E2652E41C04F014535A256755DF8D7AE069