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DESCRIBE <http://purl.uniprot.org/SHA-384/59809332A4A8FCE0262E2E19BDED0DEBF4A21A2EA68B70FFADF621B1E6E0B7063A3DD7742D097B66A6E39DE0711B8894>
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http://purl.uniprot.org/SHA-384/59809332A4A8FCE0262E2E19BDED0DEBF4A21A2EA68B70FFADF621B1E6E0B7063A3DD7742D097B66A6E39DE0711B8894
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/59809332A4A8FCE0262E2E19BDED0DEBF4A21A2EA68B70FFADF621B1E6E0B7063A3DD7742D097B66A6E39DE0711B8894
http://www.w3.org/2000/01/rdf-schema#comment
"Together these findings identify impaired disease resolution pathways in relapsing/remitting MS (RRMS) caused by dysregulated ANXA1 expression that could represent new potential therapeutic targets in RRMS."
xsd:string
http://purl.uniprot.org/uniprot/#_E10308A0D1A4491181868B354BAA6420DC37ADB70C5D766529FF05D90D8CA3886AA0A20C4FD27CC368EEFE64EA9E69D3
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/59809332A4A8FCE0262E2E19BDED0DEBF4A21A2EA68B70FFADF621B1E6E0B7063A3DD7742D097B66A6E39DE0711B8894
http://purl.uniprot.org/uniprot/Q5TZZ9
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/59809332A4A8FCE0262E2E19BDED0DEBF4A21A2EA68B70FFADF621B1E6E0B7063A3DD7742D097B66A6E39DE0711B8894
http://purl.uniprot.org/uniprot/#_Q5TZZ9-mappedCitation-31462505
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/59809332A4A8FCE0262E2E19BDED0DEBF4A21A2EA68B70FFADF621B1E6E0B7063A3DD7742D097B66A6E39DE0711B8894