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DESCRIBE <http://purl.uniprot.org/SHA-384/5AC5192491E1DADED30C05B12E9270D50D453FFE1A9CCCB3541EE6D166109A1B12C553154E1EE92EA55DB2EEAB1350E8>
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http://purl.uniprot.org/SHA-384/5AC5192491E1DADED30C05B12E9270D50D453FFE1A9CCCB3541EE6D166109A1B12C553154E1EE92EA55DB2EEAB1350E8
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/5AC5192491E1DADED30C05B12E9270D50D453FFE1A9CCCB3541EE6D166109A1B12C553154E1EE92EA55DB2EEAB1350E8
http://www.w3.org/2000/01/rdf-schema#comment
"The splice variant-derived isoforms SOM1 and SOM3 induce opposing effects in primary human endothelial cells and in a whole animal model most likely through the induction of different target genes."
xsd:string
http://purl.uniprot.org/uniprot/#_9C392167E4AD535FA9C0BC5407214D78EE984000C2DBA24DCCBD7A2BBA8F243674D61793EB82C11C8C1832F38A7FB435
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/5AC5192491E1DADED30C05B12E9270D50D453FFE1A9CCCB3541EE6D166109A1B12C553154E1EE92EA55DB2EEAB1350E8
http://purl.uniprot.org/uniprot/Q8TE85
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/5AC5192491E1DADED30C05B12E9270D50D453FFE1A9CCCB3541EE6D166109A1B12C553154E1EE92EA55DB2EEAB1350E8
http://purl.uniprot.org/uniprot/#_Q8TE85-mappedCitation-23685552
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/5AC5192491E1DADED30C05B12E9270D50D453FFE1A9CCCB3541EE6D166109A1B12C553154E1EE92EA55DB2EEAB1350E8