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DESCRIBE <http://purl.uniprot.org/SHA-384/5B6C05C380D5C79E06437978913B58058335307B79555CE31EEACDAE333C7CA3D1DD01FAB8636A199B69FF646BDD9A47>
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http://purl.uniprot.org/SHA-384/5B6C05C380D5C79E06437978913B58058335307B79555CE31EEACDAE333C7CA3D1DD01FAB8636A199B69FF646BDD9A47
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/5B6C05C380D5C79E06437978913B58058335307B79555CE31EEACDAE333C7CA3D1DD01FAB8636A199B69FF646BDD9A47
http://www.w3.org/2000/01/rdf-schema#comment
"Recent work investigating the functional consequences of MS-associated genetic variation in the gene encoding TNFR1 has demonstrated that genetic risk drives the production of a novel endogenous TNF antagonist"
xsd:string
http://purl.uniprot.org/uniprot/#_23F5CBDF2CD02FC61D05070FE4383819D95900330D13C801A7274B2AA178B45ED2721CD55E8ECD91F8465A76C6007510
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/5B6C05C380D5C79E06437978913B58058335307B79555CE31EEACDAE333C7CA3D1DD01FAB8636A199B69FF646BDD9A47
http://purl.uniprot.org/uniprot/J9PH39
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/5B6C05C380D5C79E06437978913B58058335307B79555CE31EEACDAE333C7CA3D1DD01FAB8636A199B69FF646BDD9A47
http://purl.uniprot.org/uniprot/#_J9PH39-mappedCitation-24024893
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/5B6C05C380D5C79E06437978913B58058335307B79555CE31EEACDAE333C7CA3D1DD01FAB8636A199B69FF646BDD9A47