DESCRIBE <http://purl.uniprot.org/SHA-384/5F49F94DA8F0BD127D93CE23726FF1C84D59595D8C374341DFFA69721215626036F0B295C3EECABEAD1BAA8177B18ED0>

Subject	Predicate	Object
http://purl.uniprot.org/SHA-384/5F49F94DA8F0BD127D93CE23726FF1C84D59595D8C374341DFFA69721215626036F0B295C3EECABEAD1BAA8177B18ED0	http://www.w3.org/1999/02/22-rdf-syntax-ns#type	http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/5F49F94DA8F0BD127D93CE23726FF1C84D59595D8C374341DFFA69721215626036F0B295C3EECABEAD1BAA8177B18ED0	http://www.w3.org/2000/01/rdf-schema#comment	"HDL isolated from patients with metabolic syndrome induced macrophage apoptosis oxidative stress and CHOP upregulation which were blocked by PBA and DPI. These data indicate that ox-HDL may activate ER stress-CHOP-induced apoptotic pathway in macrophages via enhanced oxidative stress and that this pathway may be mediated by TLR4."xsd:string
http://purl.uniprot.org/uniprot/#_68E87F5FA996C664C0C5F693242FA76F40C9149D4652AD9D56B17552FA188A92871F1336C6A3B95EA2BC41C9B5D07D1E	http://www.w3.org/1999/02/22-rdf-syntax-ns#subject	http://purl.uniprot.org/SHA-384/5F49F94DA8F0BD127D93CE23726FF1C84D59595D8C374341DFFA69721215626036F0B295C3EECABEAD1BAA8177B18ED0
http://purl.uniprot.org/uniprot/P35638	http://purl.uniprot.org/core/mappedAnnotation	http://purl.uniprot.org/SHA-384/5F49F94DA8F0BD127D93CE23726FF1C84D59595D8C374341DFFA69721215626036F0B295C3EECABEAD1BAA8177B18ED0
http://purl.uniprot.org/uniprot/#_P35638-mappedCitation-27895089	http://purl.uniprot.org/core/mappedAnnotation	http://purl.uniprot.org/SHA-384/5F49F94DA8F0BD127D93CE23726FF1C84D59595D8C374341DFFA69721215626036F0B295C3EECABEAD1BAA8177B18ED0