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DESCRIBE <http://purl.uniprot.org/SHA-384/5FDE919311589AA44A6C598C2037E759462A6CE5CBBE65ED042739ADC7D63E6F0593D496FE5B26A149EF9F5E29B3F6D9>
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http://purl.uniprot.org/SHA-384/5FDE919311589AA44A6C598C2037E759462A6CE5CBBE65ED042739ADC7D63E6F0593D496FE5B26A149EF9F5E29B3F6D9
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/5FDE919311589AA44A6C598C2037E759462A6CE5CBBE65ED042739ADC7D63E6F0593D496FE5B26A149EF9F5E29B3F6D9
http://www.w3.org/2000/01/rdf-schema#comment
"These findings suggest that ezrin-EGFR interaction augments oncogenic functions of EGFR and that targeting ezrin may provide a potential novel approach to overcome erlotinib resistance in non-small cell lung cancer cells"
xsd:string
http://purl.uniprot.org/uniprot/#_48F3C3D1C4D1A5DED017561C5CC090F92623B197BCBA73789E21B180B3955F272D60509BE736F35673E6B6FE1F17BDA9
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/5FDE919311589AA44A6C598C2037E759462A6CE5CBBE65ED042739ADC7D63E6F0593D496FE5B26A149EF9F5E29B3F6D9
http://purl.uniprot.org/uniprot/V9HW42
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/5FDE919311589AA44A6C598C2037E759462A6CE5CBBE65ED042739ADC7D63E6F0593D496FE5B26A149EF9F5E29B3F6D9
http://purl.uniprot.org/uniprot/#_V9HW42-mappedCitation-26936397
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/5FDE919311589AA44A6C598C2037E759462A6CE5CBBE65ED042739ADC7D63E6F0593D496FE5B26A149EF9F5E29B3F6D9