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DESCRIBE <http://purl.uniprot.org/SHA-384/60AFCE0F2463FE2E344B77210514330E52BEDF2B3AB27099FC13671DC0EA9EC41A373C0129F28C01351A892FF1B6924E>
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http://purl.uniprot.org/SHA-384/60AFCE0F2463FE2E344B77210514330E52BEDF2B3AB27099FC13671DC0EA9EC41A373C0129F28C01351A892FF1B6924E
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/60AFCE0F2463FE2E344B77210514330E52BEDF2B3AB27099FC13671DC0EA9EC41A373C0129F28C01351A892FF1B6924E
http://www.w3.org/2000/01/rdf-schema#comment
"We hypothesize that SOCS1 functions as suppressor of IFN-gamma signaling not only by inhibiting STAT1 activation but also by sustaining ERK1/2-dependent antiinflammatory pathways."
xsd:string
http://purl.uniprot.org/uniprot/#_52463810F8A326B3656AA6CB2227351D9B1BF6B9D3EF9045F3E4A65C21E25898127455E15BC7A09C241663D2E98EB6EB
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/60AFCE0F2463FE2E344B77210514330E52BEDF2B3AB27099FC13671DC0EA9EC41A373C0129F28C01351A892FF1B6924E
http://purl.uniprot.org/uniprot/Q96LA2
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/60AFCE0F2463FE2E344B77210514330E52BEDF2B3AB27099FC13671DC0EA9EC41A373C0129F28C01351A892FF1B6924E
http://purl.uniprot.org/uniprot/#_Q96LA2-mappedCitation-18556463
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/60AFCE0F2463FE2E344B77210514330E52BEDF2B3AB27099FC13671DC0EA9EC41A373C0129F28C01351A892FF1B6924E