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DESCRIBE <http://purl.uniprot.org/SHA-384/64C144E19549556FBFB44C26E958B75ADCDDC23C14142B54C43B29B4B836845924CE79F0103D2DB5140067ADFF322A93>
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http://purl.uniprot.org/SHA-384/64C144E19549556FBFB44C26E958B75ADCDDC23C14142B54C43B29B4B836845924CE79F0103D2DB5140067ADFF322A93
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/64C144E19549556FBFB44C26E958B75ADCDDC23C14142B54C43B29B4B836845924CE79F0103D2DB5140067ADFF322A93
http://www.w3.org/2000/01/rdf-schema#comment
"Our results demonstrated that MSI2 maintained the stemness of CD44v6+ LCSCs by activating Notch1 signaling through the interaction with LFNG which could be a potential molecular target for stem cell-targeted therapy for liver cancer."
xsd:string
http://purl.uniprot.org/uniprot/#_979DC0B5D5C3C31B73E195FA5B5A26A60BA4DF5AA25C88B5C70731B2CDAFCC313BFCCAE1443D3C711FCA9626F70F4A41
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/64C144E19549556FBFB44C26E958B75ADCDDC23C14142B54C43B29B4B836845924CE79F0103D2DB5140067ADFF322A93
http://purl.uniprot.org/uniprot/B4DHE8
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/64C144E19549556FBFB44C26E958B75ADCDDC23C14142B54C43B29B4B836845924CE79F0103D2DB5140067ADFF322A93
http://purl.uniprot.org/uniprot/#_B4DHE8-mappedCitation-31888685
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/64C144E19549556FBFB44C26E958B75ADCDDC23C14142B54C43B29B4B836845924CE79F0103D2DB5140067ADFF322A93