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DESCRIBE <http://purl.uniprot.org/SHA-384/668ACFCCE9DBD5F1D5C8B3E23FE5BB7B65E9291007B1B36AEA97DA44D0F8DE7B11985D0D4B6CD9E6CD10F9D9FF47B9F3>
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http://purl.uniprot.org/SHA-384/668ACFCCE9DBD5F1D5C8B3E23FE5BB7B65E9291007B1B36AEA97DA44D0F8DE7B11985D0D4B6CD9E6CD10F9D9FF47B9F3
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/668ACFCCE9DBD5F1D5C8B3E23FE5BB7B65E9291007B1B36AEA97DA44D0F8DE7B11985D0D4B6CD9E6CD10F9D9FF47B9F3
http://www.w3.org/2000/01/rdf-schema#comment
"Homozygous Cln3(delta ex7/8) transgenic mice represent the most appropriate disease model for studying the development of the pathogenetic events of juvenile neuronal ceroidlipofuscinoses."
xsd:string
http://purl.uniprot.org/uniprot/#_FA7DF359055A5A07EBD5B0158C85AAD6EDC79CB96A91571F53C06CD46A52C79179F4DC9AAD743D293157C9F8FC59A76A
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/668ACFCCE9DBD5F1D5C8B3E23FE5BB7B65E9291007B1B36AEA97DA44D0F8DE7B11985D0D4B6CD9E6CD10F9D9FF47B9F3
http://purl.uniprot.org/uniprot/Q5XFT9
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/668ACFCCE9DBD5F1D5C8B3E23FE5BB7B65E9291007B1B36AEA97DA44D0F8DE7B11985D0D4B6CD9E6CD10F9D9FF47B9F3
http://purl.uniprot.org/uniprot/#_Q5XFT9-mappedCitation-18265413
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/668ACFCCE9DBD5F1D5C8B3E23FE5BB7B65E9291007B1B36AEA97DA44D0F8DE7B11985D0D4B6CD9E6CD10F9D9FF47B9F3