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http://purl.uniprot.org/SHA-384/67CD031F5BB19C57E0EDAA5F8A49365ED9B5C3840A0DB2B54532B81B29E295130049E0B106263B108A475BAB4F49F405http://www.w3.org/1999/02/22-rdf-syntax-ns#typehttp://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/67CD031F5BB19C57E0EDAA5F8A49365ED9B5C3840A0DB2B54532B81B29E295130049E0B106263B108A475BAB4F49F405http://www.w3.org/2000/01/rdf-schema#comment"torsinA loss elevates LINC complex levels in the proliferative zone and that genetic reduction of LINC complexes prevents abnormal brain morphogenesis in Tor1a-/- embryos. These data show that Tor1a affects radial glial cells via a LINC complex mediated mechanism. They also predict human TOR1A disease will include incompletely penetrant defects in embryonic brain morphogenesis in cases where mutations ablate TOR1A"xsd:string
http://purl.uniprot.org/uniprot/#_35208981776107EE4FB06CA5D3A418DCD2667D5AF14C35249A8291C2359C784F3FC2D4D8D0F313A58EC1681F06B347D3http://www.w3.org/1999/02/22-rdf-syntax-ns#subjecthttp://purl.uniprot.org/SHA-384/67CD031F5BB19C57E0EDAA5F8A49365ED9B5C3840A0DB2B54532B81B29E295130049E0B106263B108A475BAB4F49F405
http://purl.uniprot.org/uniprot/B3KPA1http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/67CD031F5BB19C57E0EDAA5F8A49365ED9B5C3840A0DB2B54532B81B29E295130049E0B106263B108A475BAB4F49F405
http://purl.uniprot.org/uniprot/#_B3KPA1-mappedCitation-29868845http://purl.uniprot.org/core/mappedAnnotationhttp://purl.uniprot.org/SHA-384/67CD031F5BB19C57E0EDAA5F8A49365ED9B5C3840A0DB2B54532B81B29E295130049E0B106263B108A475BAB4F49F405