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DESCRIBE <http://purl.uniprot.org/SHA-384/688E85DE1102626FAD7E01A3CC5FB4519722933E54B0D29CDB0344F4AF565C025DB813FE0BDD908D5CC95E1890743BD9>
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http://purl.uniprot.org/SHA-384/688E85DE1102626FAD7E01A3CC5FB4519722933E54B0D29CDB0344F4AF565C025DB813FE0BDD908D5CC95E1890743BD9
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/688E85DE1102626FAD7E01A3CC5FB4519722933E54B0D29CDB0344F4AF565C025DB813FE0BDD908D5CC95E1890743BD9
http://www.w3.org/2000/01/rdf-schema#comment
"This study not only further supported the important role of GPD1L in Cardiac conduction disease but also expanded the spectrum of GPD1L mutations"
xsd:string
http://purl.uniprot.org/uniprot/#_BB304608537153BD8C637CC092BF4C20498EAB64CE8EE0699348DFFC085B06440310E8270EBEC7B1E999A5C1F22313AE
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/688E85DE1102626FAD7E01A3CC5FB4519722933E54B0D29CDB0344F4AF565C025DB813FE0BDD908D5CC95E1890743BD9
http://purl.uniprot.org/uniprot/B3KWN2
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/688E85DE1102626FAD7E01A3CC5FB4519722933E54B0D29CDB0344F4AF565C025DB813FE0BDD908D5CC95E1890743BD9
http://purl.uniprot.org/uniprot/#_B3KWN2-mappedCitation-29077258
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/688E85DE1102626FAD7E01A3CC5FB4519722933E54B0D29CDB0344F4AF565C025DB813FE0BDD908D5CC95E1890743BD9