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DESCRIBE <http://purl.uniprot.org/SHA-384/6A668C4AA487D08F3F568C76C7DE9D8AE12FF9B6C143D26DBE6B7DC899B9B8AC26F9C558FB9E97DB23ADBD04C686FA3C>
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http://purl.uniprot.org/SHA-384/6A668C4AA487D08F3F568C76C7DE9D8AE12FF9B6C143D26DBE6B7DC899B9B8AC26F9C558FB9E97DB23ADBD04C686FA3C
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/6A668C4AA487D08F3F568C76C7DE9D8AE12FF9B6C143D26DBE6B7DC899B9B8AC26F9C558FB9E97DB23ADBD04C686FA3C
http://www.w3.org/2000/01/rdf-schema#comment
"The increased expression of dysbindin might play a role in the pathogenesis of drug-refractory temporal lobe epilepsy."
xsd:string
http://purl.uniprot.org/uniprot/#_D71AA74C015A6374CAAEAB037C8701105E7714FA401DFAE4A0580B7D2E4F0138593798613E1D0872FCD6AB2EC6974AB8
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/6A668C4AA487D08F3F568C76C7DE9D8AE12FF9B6C143D26DBE6B7DC899B9B8AC26F9C558FB9E97DB23ADBD04C686FA3C
http://purl.uniprot.org/uniprot/Q96EV8
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6A668C4AA487D08F3F568C76C7DE9D8AE12FF9B6C143D26DBE6B7DC899B9B8AC26F9C558FB9E97DB23ADBD04C686FA3C
http://purl.uniprot.org/uniprot/#_Q96EV8-mappedCitation-22337344
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6A668C4AA487D08F3F568C76C7DE9D8AE12FF9B6C143D26DBE6B7DC899B9B8AC26F9C558FB9E97DB23ADBD04C686FA3C