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DESCRIBE <http://purl.uniprot.org/SHA-384/6B409E417B2EC1B6A4B9A97424507C5F8B953597E38ECAEB130DFC3D590DA23C36DC1DE0D274432B0526A3284E430784>
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http://purl.uniprot.org/SHA-384/6B409E417B2EC1B6A4B9A97424507C5F8B953597E38ECAEB130DFC3D590DA23C36DC1DE0D274432B0526A3284E430784
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/6B409E417B2EC1B6A4B9A97424507C5F8B953597E38ECAEB130DFC3D590DA23C36DC1DE0D274432B0526A3284E430784
http://www.w3.org/2000/01/rdf-schema#comment
"Aldosterone-induced upregulation of NCC Sgk1 and ENaC likely contributes to the antinatriuretic phase of metabolic acidosis. This adaptation prevents Na(+) wasting and volume depletion during chronic acid insult."
xsd:string
http://purl.uniprot.org/uniprot/#_89A828A60F52C0342409BCBCAECCA2B07CA6D2DC8B46B8E54B1A23F2A5E0EE0FD234980C8505209958C7B9337BC7DB91
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/6B409E417B2EC1B6A4B9A97424507C5F8B953597E38ECAEB130DFC3D590DA23C36DC1DE0D274432B0526A3284E430784
http://purl.uniprot.org/uniprot/P55018
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6B409E417B2EC1B6A4B9A97424507C5F8B953597E38ECAEB130DFC3D590DA23C36DC1DE0D274432B0526A3284E430784
http://purl.uniprot.org/uniprot/#_P55018-mappedCitation-16495212
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6B409E417B2EC1B6A4B9A97424507C5F8B953597E38ECAEB130DFC3D590DA23C36DC1DE0D274432B0526A3284E430784