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DESCRIBE <http://purl.uniprot.org/SHA-384/6B6BA28F82D6E6DB31D60D1FB41BEC2D671B7EDEF4C4F62EC0A9D1833268D78654B0E4B5754627B044804418CC0DCB4A>
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http://purl.uniprot.org/SHA-384/6B6BA28F82D6E6DB31D60D1FB41BEC2D671B7EDEF4C4F62EC0A9D1833268D78654B0E4B5754627B044804418CC0DCB4A
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/6B6BA28F82D6E6DB31D60D1FB41BEC2D671B7EDEF4C4F62EC0A9D1833268D78654B0E4B5754627B044804418CC0DCB4A
http://www.w3.org/2000/01/rdf-schema#comment
"Under conditions of chronic hemodynamic stress enhanced MAO-B activity is a major determinant of cardiac structural and functional disarrangement."
xsd:string
http://purl.uniprot.org/uniprot/#_F6890CB9C2238858DDE313B8E064568B81AB34883CFEE63DDA2A77715AEB2EF8249D18DBCBF3AD132F15319E4939D498
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/6B6BA28F82D6E6DB31D60D1FB41BEC2D671B7EDEF4C4F62EC0A9D1833268D78654B0E4B5754627B044804418CC0DCB4A
http://purl.uniprot.org/uniprot/Q3UJ53
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6B6BA28F82D6E6DB31D60D1FB41BEC2D671B7EDEF4C4F62EC0A9D1833268D78654B0E4B5754627B044804418CC0DCB4A
http://purl.uniprot.org/uniprot/#_Q3UJ53-mappedCitation-23581564
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6B6BA28F82D6E6DB31D60D1FB41BEC2D671B7EDEF4C4F62EC0A9D1833268D78654B0E4B5754627B044804418CC0DCB4A