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DESCRIBE <http://purl.uniprot.org/SHA-384/6B9507F379B28988E62DCEA22759651ED281B8DB58A91F7285F5C5E1E984B930908D94961BFDA395CD5B88D237ADCB39>
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http://purl.uniprot.org/SHA-384/6B9507F379B28988E62DCEA22759651ED281B8DB58A91F7285F5C5E1E984B930908D94961BFDA395CD5B88D237ADCB39
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/6B9507F379B28988E62DCEA22759651ED281B8DB58A91F7285F5C5E1E984B930908D94961BFDA395CD5B88D237ADCB39
http://www.w3.org/2000/01/rdf-schema#comment
"results suggest that secretion of IGF-I from ATII cells results in autocrine activation of Wnt5a signaling that promotes both wound repair and differentiation"
xsd:string
http://purl.uniprot.org/uniprot/#_145EEAEE33864F2DE5D3EB95F6FDB2205312A9519FEE286F02647E19DD5E746E4000A5858988DE57410A03E2BC9E4464
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/6B9507F379B28988E62DCEA22759651ED281B8DB58A91F7285F5C5E1E984B930908D94961BFDA395CD5B88D237ADCB39
http://purl.uniprot.org/uniprot/Q5RK13
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6B9507F379B28988E62DCEA22759651ED281B8DB58A91F7285F5C5E1E984B930908D94961BFDA395CD5B88D237ADCB39
http://purl.uniprot.org/uniprot/#_Q5RK13-mappedCitation-23709620
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6B9507F379B28988E62DCEA22759651ED281B8DB58A91F7285F5C5E1E984B930908D94961BFDA395CD5B88D237ADCB39