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DESCRIBE <http://purl.uniprot.org/SHA-384/6C0FA99B277A55C5209CEB9E2665F6B1EC95E6F7A21F7533FF55D69B6CFF364C92AA337C20AC62FEAE2711B33C03D93A>
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http://purl.uniprot.org/SHA-384/6C0FA99B277A55C5209CEB9E2665F6B1EC95E6F7A21F7533FF55D69B6CFF364C92AA337C20AC62FEAE2711B33C03D93A
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/6C0FA99B277A55C5209CEB9E2665F6B1EC95E6F7A21F7533FF55D69B6CFF364C92AA337C20AC62FEAE2711B33C03D93A
http://www.w3.org/2000/01/rdf-schema#comment
"Results confirm earlier findings that the MC4R-Kir7.1 signaling is independent of Gs-AC-cAMP signaling pathway. Furthermore these data suggest that a noncanonical GPCR signaling pathway may be essential for this interaction."
xsd:string
http://purl.uniprot.org/uniprot/#_5CBCD476BD81E8E8C19D57238828C535086E4D2B858104D960816A14D3128A703A3B25720555ABCED67F1E04399A154F
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/6C0FA99B277A55C5209CEB9E2665F6B1EC95E6F7A21F7533FF55D69B6CFF364C92AA337C20AC62FEAE2711B33C03D93A
http://purl.uniprot.org/uniprot/O60928
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6C0FA99B277A55C5209CEB9E2665F6B1EC95E6F7A21F7533FF55D69B6CFF364C92AA337C20AC62FEAE2711B33C03D93A
http://purl.uniprot.org/uniprot/#_O60928-mappedCitation-29058194
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6C0FA99B277A55C5209CEB9E2665F6B1EC95E6F7A21F7533FF55D69B6CFF364C92AA337C20AC62FEAE2711B33C03D93A