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DESCRIBE <http://purl.uniprot.org/SHA-384/6C6FE392241B5E351E045290F383BAFA996CDF6639D11BF8C886E620C65B2C965A91BEE21F65B473B02769C777503A64>
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http://purl.uniprot.org/SHA-384/6C6FE392241B5E351E045290F383BAFA996CDF6639D11BF8C886E620C65B2C965A91BEE21F65B473B02769C777503A64
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/6C6FE392241B5E351E045290F383BAFA996CDF6639D11BF8C886E620C65B2C965A91BEE21F65B473B02769C777503A64
http://www.w3.org/2000/01/rdf-schema#comment
"global null deletion of Ceacam1 caused an increase in blood pressure with increased renin-angiotensin system activation together with upregulation of prorenin receptor via PI3K-Akt activation of CREB-1 ATF-1 ATF-2 and NF-kappaB p65."
xsd:string
http://purl.uniprot.org/uniprot/#_D5FC3492C55E9DEDB04A490F6E173617A876BB1A6BBBB49F7DD5B60284E9FFB73634BFEE3BDFD0CF2C4D843385916B68
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/6C6FE392241B5E351E045290F383BAFA996CDF6639D11BF8C886E620C65B2C965A91BEE21F65B473B02769C777503A64
http://purl.uniprot.org/uniprot/Q3LFS9
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6C6FE392241B5E351E045290F383BAFA996CDF6639D11BF8C886E620C65B2C965A91BEE21F65B473B02769C777503A64
http://purl.uniprot.org/uniprot/#_Q3LFS9-mappedCitation-23734002
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6C6FE392241B5E351E045290F383BAFA996CDF6639D11BF8C886E620C65B2C965A91BEE21F65B473B02769C777503A64