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DESCRIBE <http://purl.uniprot.org/SHA-384/6E27464134C2394D66D10867C3B4CF721E17585E6E0FB84A0287DCA6BACF77A3862C317794D9E92A15C2183E758D38D6>
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http://purl.uniprot.org/SHA-384/6E27464134C2394D66D10867C3B4CF721E17585E6E0FB84A0287DCA6BACF77A3862C317794D9E92A15C2183E758D38D6
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/6E27464134C2394D66D10867C3B4CF721E17585E6E0FB84A0287DCA6BACF77A3862C317794D9E92A15C2183E758D38D6
http://www.w3.org/2000/01/rdf-schema#comment
"define the first physiologic function for Noxa and suggest that by repressing Noxa induction of G arrest by p18INK4c bypasses a homeostatic cell-cycle checkpoint in intermediate plasma cells (iPCs) for PC differentiation"
xsd:string
http://purl.uniprot.org/uniprot/#_97653E049C31FBD0051455D97EAE9F2BF472B32B015DAF3A9C117E2F289B80791B13F39A09F168105899C41CEAEB6C5F
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/6E27464134C2394D66D10867C3B4CF721E17585E6E0FB84A0287DCA6BACF77A3862C317794D9E92A15C2183E758D38D6
http://purl.uniprot.org/uniprot/Q9D153
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6E27464134C2394D66D10867C3B4CF721E17585E6E0FB84A0287DCA6BACF77A3862C317794D9E92A15C2183E758D38D6
http://purl.uniprot.org/uniprot/#_Q9D153-mappedCitation-21163929
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6E27464134C2394D66D10867C3B4CF721E17585E6E0FB84A0287DCA6BACF77A3862C317794D9E92A15C2183E758D38D6