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DESCRIBE <http://purl.uniprot.org/SHA-384/6F7B769BAFC4D54405AD8C3397778410AE80FB9995F6BF6EF8EA11EE908B0D6F9791A8A85E2E0687DE4F04B1ECDFAA32>
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http://purl.uniprot.org/SHA-384/6F7B769BAFC4D54405AD8C3397778410AE80FB9995F6BF6EF8EA11EE908B0D6F9791A8A85E2E0687DE4F04B1ECDFAA32
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/6F7B769BAFC4D54405AD8C3397778410AE80FB9995F6BF6EF8EA11EE908B0D6F9791A8A85E2E0687DE4F04B1ECDFAA32
http://www.w3.org/2000/01/rdf-schema#comment
"Study showed that IGFBP3 is dramatically induced in pancreatic tumors and is abundantly produced in pancreatic cancer cells causing muscle wasting through both impaired myogenesis via at least inhibition of IGF/PI3K/AKT signaling."
xsd:string
http://purl.uniprot.org/uniprot/#_2DD91FC0D3A78A92043444BF47865820AC6EFC51C7906BC84CD64AADFE74CC42D5AFF44779309D13FB8CB17F29BA9C45
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/6F7B769BAFC4D54405AD8C3397778410AE80FB9995F6BF6EF8EA11EE908B0D6F9791A8A85E2E0687DE4F04B1ECDFAA32
http://purl.uniprot.org/uniprot/B3KTQ0
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6F7B769BAFC4D54405AD8C3397778410AE80FB9995F6BF6EF8EA11EE908B0D6F9791A8A85E2E0687DE4F04B1ECDFAA32
http://purl.uniprot.org/uniprot/#_B3KTQ0-mappedCitation-26975989
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/6F7B769BAFC4D54405AD8C3397778410AE80FB9995F6BF6EF8EA11EE908B0D6F9791A8A85E2E0687DE4F04B1ECDFAA32