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DESCRIBE <http://purl.uniprot.org/SHA-384/71EA0ECAB86A1E18F5BB32890D4C41951D73FCD29FD2FD0E2D60C8F4DEBE142B9B945C2C7E2934AC2DEBF9B874C16F0E>
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http://purl.uniprot.org/SHA-384/71EA0ECAB86A1E18F5BB32890D4C41951D73FCD29FD2FD0E2D60C8F4DEBE142B9B945C2C7E2934AC2DEBF9B874C16F0E
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/71EA0ECAB86A1E18F5BB32890D4C41951D73FCD29FD2FD0E2D60C8F4DEBE142B9B945C2C7E2934AC2DEBF9B874C16F0E
http://www.w3.org/2000/01/rdf-schema#comment
"EAAC1 is the primary route for neuronal cysteine uptake and EAAC1 deficiency leads to impaired neuronal glutathione metabolism"
xsd:string
http://purl.uniprot.org/uniprot/#_A8F8F9E6A6BD84C6E4E822C642AA0308155441ECBAB8195E51BB1697E932F4D47F11D4F7FAD87FCD7CC9F25658082205
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/71EA0ECAB86A1E18F5BB32890D4C41951D73FCD29FD2FD0E2D60C8F4DEBE142B9B945C2C7E2934AC2DEBF9B874C16F0E
http://purl.uniprot.org/uniprot/Q8VI14
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/71EA0ECAB86A1E18F5BB32890D4C41951D73FCD29FD2FD0E2D60C8F4DEBE142B9B945C2C7E2934AC2DEBF9B874C16F0E
http://purl.uniprot.org/uniprot/#_Q8VI14-mappedCitation-16311588
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/71EA0ECAB86A1E18F5BB32890D4C41951D73FCD29FD2FD0E2D60C8F4DEBE142B9B945C2C7E2934AC2DEBF9B874C16F0E