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DESCRIBE <http://purl.uniprot.org/SHA-384/72D4F9BAFF216D57A398E530950C8393A5804F5CB51FB7E08B486D522C776CADCD1A06536092BE87DA8F2996F983740D>
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http://purl.uniprot.org/SHA-384/72D4F9BAFF216D57A398E530950C8393A5804F5CB51FB7E08B486D522C776CADCD1A06536092BE87DA8F2996F983740D
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/72D4F9BAFF216D57A398E530950C8393A5804F5CB51FB7E08B486D522C776CADCD1A06536092BE87DA8F2996F983740D
http://www.w3.org/2000/01/rdf-schema#comment
"This study revealed that FOXP1-induced CLRN1-AS1 regulated cellular functions in pituitary prolactinoma by sponging miR-217 to release the DKK1/Wnt/beta-catenin signaling pathway."
xsd:string
http://purl.uniprot.org/uniprot/#_1B3117B6B186A7154FAE7F232AF00DED7151B39B9E41BBE565398C417387D87DD0B8C951D328C2A56E031C02A5A4E03C
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/72D4F9BAFF216D57A398E530950C8393A5804F5CB51FB7E08B486D522C776CADCD1A06536092BE87DA8F2996F983740D
http://purl.uniprot.org/uniprot/Q8CCZ5
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/72D4F9BAFF216D57A398E530950C8393A5804F5CB51FB7E08B486D522C776CADCD1A06536092BE87DA8F2996F983740D
http://purl.uniprot.org/uniprot/#_Q8CCZ5-mappedCitation-31235696
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/72D4F9BAFF216D57A398E530950C8393A5804F5CB51FB7E08B486D522C776CADCD1A06536092BE87DA8F2996F983740D