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DESCRIBE <http://purl.uniprot.org/SHA-384/730764AB796ACE1B2F6A5F7B10B6D1856061183C35F6A60A9637A4965E102D79016511608B9242188B74EAE0DCB03507>
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http://purl.uniprot.org/SHA-384/730764AB796ACE1B2F6A5F7B10B6D1856061183C35F6A60A9637A4965E102D79016511608B9242188B74EAE0DCB03507
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://purl.uniprot.org/core/Annotation
http://purl.uniprot.org/SHA-384/730764AB796ACE1B2F6A5F7B10B6D1856061183C35F6A60A9637A4965E102D79016511608B9242188B74EAE0DCB03507
http://www.w3.org/2000/01/rdf-schema#comment
"increase in KCa3.1 activity induced by the upregulation of KCa3.1a and NDPK-B may be involved in the pathogenesis of IBD by mediating the enhancement of the proliferative response in MLN CD4(+) T lymphocyte."
xsd:string
http://purl.uniprot.org/uniprot/#_BC0D048FC15AD56E49A3EE681EC1909FB1C3A70933B47330A8103989C2062DA453EAFD175AE9F805BCDC5F0FBDD2FAA4
http://www.w3.org/1999/02/22-rdf-syntax-ns#subject
http://purl.uniprot.org/SHA-384/730764AB796ACE1B2F6A5F7B10B6D1856061183C35F6A60A9637A4965E102D79016511608B9242188B74EAE0DCB03507
http://purl.uniprot.org/uniprot/Q01768
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/730764AB796ACE1B2F6A5F7B10B6D1856061183C35F6A60A9637A4965E102D79016511608B9242188B74EAE0DCB03507
http://purl.uniprot.org/uniprot/#_Q01768-mappedCitation-24674776
http://purl.uniprot.org/core/mappedAnnotation
http://purl.uniprot.org/SHA-384/730764AB796ACE1B2F6A5F7B10B6D1856061183C35F6A60A9637A4965E102D79016511608B9242188B74EAE0DCB03507